Chapter 12: Kidney
Let's not kid(ney) ourselves: renal physiology is tough! Acute tubular injury (ATI) is a common cause of acute kidney injury (AKI) and AKI is a significant cause of morbidity and mortality, particularly in hospitalized patients. ATI often gets short shrift in the medical curriculum because there is such a fuss about glomerulonephritis (which is also quite tough! check out the Pathology Central video on Nephrotic and Nephritic syndromes) and renal tumors. This short video will help you catch up on the topic and be prepared for the wards and the Boards!
Don't just memorize the features of nephritic (hematuria, mild/moderate proteinuria/azotemia/hypertension) and nephrotic (massive proteinuria, hypoalbuminemia, generalized edema, hyperlipidemia/lipiduria) syndrome. UNDERSTAND the pathophysiology! Deeper understanding = better medicine. This video compares and contrasts the pathophysiology and clinical findings of nephritic and nephrotic syndrome and analyzes a classic example of each.
This is a short jaunt through the major benign (oncocytoma, angiomyolipoma) and malignant (clear cell renal cell carcinoma, papillary renal cell carcinoma and chromophobe renal cell carcinoma). We'll talk about pathophysiology, look at some great pictures and do some compare and contrast. This video is for medical students, though there are some nice tidbits for more advanced study as well.